O2-Suppl increases exercise-tolerance, resulting in increased muscle injury markers in COPD. The O2-Suppl protocol resulted in a lower increase in CRP (92.1 ± 112.4% vs. CPK, LDH and CRP were measured before, immediately after and 24 hours after EET.Įxercise time was higher with O2-Suppl (49.9 ± 37.3% p = 0.001) and increases in CPK and LDH were observed compared to basal values in the O2-Suppl (28.4UI/L and 28.3 UI/L).
We aimed to investigate the effect of O2-Suppl during exercise test (EET) on CRP level and muscle damage (CPK, LDH, lactate) in non-hypoxemic COPD patients.Įleven non-depleted patients with COPD (FEV1 65.5 ± 4.3%) performed two EET (room-air or O2-Suppl-100%), through a blind, randomized, and placebo-controlled crossover design. However, high-intensity exercise tends toward muscle damage and a greater inflammatory response. Oxygen supplementation (O2-Suppl) is recommended for pulmonary rehabilitation with higher exercise intensities. However, the achieved values reflect a normal response to a moderate-intensity exercise. Conclusion: Both the low-frequency NMES (30 Hz) and the high-frequency (100 Hz) elevate blood markers of muscle damage, most strikingly at the higher frequency. The perception of contractile efficiency decreased significantly (p = 0.016) in the 100 Hz Protocol. 100 Hz the following were observed: lactate (23.7 ± 6.7 vs.
Measured lactate, creatine phosphokinase and lactate dehydrogenase before, immediately after, and 6 and 48 hours after the protocols. (A)-20 minutes of NMES in the quadriceps at a frequency of 30 Hz (B)-20 minutes of NMES at a frequency of 100 Hz. Two protocols (A) and (B) with an interval of 7 days between them. The study included 10 male college students, age 24.4 ± 6.0 years, weight 77.1 ± 11.8 kg, height 176.1 ± 5.6 cm, and BMI of 24.8 ± 3 4 kg/m 2. Methods: Experimental crossover study, randomized, unblinded. Objective: To evaluate the muscle damage stemming from isometric muscle contraction induced by NMES of low frequency (30 Hz) and high frequency (100 Hz). Neuromuscular electrical stimulation (NMES) is the action of therapeutic electrical stimulation on muscle tissue in order to contract the muscle and consequently improve the muscle status.
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